The most effective natural Anti-Aging treatment |
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| WOMEN 39 AND UNDER | MEN 39 AND UNDER | WOMEN 40 AND UP | MEN 40 AND UP | |||
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What is Aging?
"Aging" is the internationally recognised term for the aging process and also a general term for the reason we develop more than 300 of the most commonly occurring diseases. So when we talk about an anti-aging treatment, we are not just referring to an anti-wrinkle treatment but one that also covers the ground for most diseases.
Stress, smoking, pollution and chronic inflammation, among other things, speeds up the aging process significantly. Today, we know that we can influence the speed of the aging process and thereby reduce our risk of becoming sick and suffering from premature death. Essential natural antioxidants and anti-inflammatory substances (see below) help to strengthen our immune system and reduce our risk of unnecessary premature disease and death.
Which natural ingredients can have an effect on aging?
Carotenoids, alpha-lipoic acid, vitamins C and E, coenzyme Q10, ginkgo biloba, grape seed extract, green tea extract, N-acetylcysteine, tocotrienols and many more.
What effects do these ingredients have?
- Slow down the aging process.
- Reduce fine wrinkes and skin roughness by 21.2%.
- Improve overall well-being.
- Reduce the risk of developing many chronic diseases.
- Decrease the risk of premature death.
- Speed up the healing process caused by sports injuries and other tissue related injuries.
For the reader with a deeper interest:
How do these ingredients work?
- Treat and prevent vitamin and mineral deficiencies.
- Inhibit oxidative stress on several levels.
- Significant anti-inflammatory effect on several levels.
- Prevent brain cell oxidation.
- Boost the activity of certain immune system cells.
- Block the platelet-activating factor (PAF).
- Inhibit the development of cancer cells on several levels.
- Inhibit the spread of cancer cells.
- Prevent the development of glaucoma.
- Inhibit the release of histamine.
- Lower blood cholesterol by reverting cholesterol transport.
- Stabilise the capillary wall.
- Prevent the depletion of glutathione.
- Reduce the impact of heart attack and ventricle function.
- Neutralise the toxic effect of mercury and other heavy metals.
- Normalise the constitution of the intestinal flora.
The information above is based on more than 9,000 articles published in scientific journals and which are available at MEDLINE, the world’s largest medical database. Below you will find a few of them listed:
References
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- Bliznakov EG et al. Biochemical and clinical consequences of inhibiting coenzyme Q(10) biosynthesis by lipid-lowering HMG-CoA reductase inhibitors (statins): a critical overview. 1998. Adv Ther 15: 218-228.
- Dlugosz A et al. The chemoprotective effect of coenzyme Q on lipids in the paint and lacquer industry workers. 1998. Intl J Occup Med Env Health 11: 153-163.
- Ernster L et al. Biochemical, physiological and medical aspects of ubiquinone function. 1995. Biochim Biophys Acta 1271: 195-204.
- Ferrara N et al. Protective role of chronic ubiquinone administration on acute cardiac oxidative stress. 1995. J Pharmacol Exp Ther 274: 858-865.
- Folkers K et al. A one year bioavailability study of coenzyme Q10 with 3 months withdrawal period. 1994. Molec Aspects Med 15, Suppl: S281-285.
- Folkers K et al. Biochemical rationale and myocardial tissue data on the effective therapy of cardiomyopathy with coenzyme Q10. 1985. Proc Natl Acad Sci USA 82: 901-904.
- Forsmark-Andree P et al. Evidence for a protective effect of endogenous ubiquinol against oxidative damage to mitochondrial protein and DNA during lipid peroxidation. 1994. Molec Aspects Med 15, Suppl: S73-S81.
- Forsmark-Andree P et al. Lipid peroxidation and changes in the ubiquinone content and the respiratory chain enzymes of submitochondrial particles. 1997. Free Radic Biol Med 22: 391-400.
- Halliwell B et al. Free radicals in biology and medicine, 3rd edn. Oxford, 1999.
- Hanaki Y et al. Coenzyme Q10 and coronary artery disease. 1993. Clin Investig 71, Suppl: S112-S115.
- Harman D. Aging: A theory based on free radical and radiation chemistry. 1956. J Gerontol 12: 298-300.
- Harman D. Extending functional life span. 1998. Exp Gerontol 33: 95-112.
- Harman D. The biological clock: the mitochondria? 1972. J Am Geriatr Soc 20: 145-147.
- Herrero A, Barja G. Effect of aging on mitochondrial and nuclear DNA oxidative damage in the heart and brain throughout the life-span of the rat. J Amer Aging Assoc. 2001 Apr;24(2):45-50.
- Humphries KM et al. Inhibition of NADH-linked mitochondrial respiration by 4-hydroxy-2-nonenal. 1998. Biochemistry 37: 552-557.
- Kamikawa T et al. Effects of coenzyme Q10 on exercise tolerance in chronic stable angina pectoris. 1985. Am J Cardiol 56: 247-251.
- Kontush A et al. Antioxidative activity of ubiquinol-10 at physiologic concentrations in human low density lipoprotein. 1995. Biochem Biophys Acta 1258: 177-187.
- Langsjoen H et al. Usefulness of coenzyme Q10 in clinical cardiology: a long-term study. 1994. Molec Aspects Med 15, Suppl: S165-S175.
- Langsjoen P et al. Treatment of essential hypertension with coenzyme Q10. 1994. Molec Aspects Med 15, Suppl: S265-S272.
- Langsjoen PH et al. The aging heart: reversal of diastolic dysfunction through the use of oral CoQ10 in the elderly. In Klatz RM et al. (eds), Anti-Aging Medical Therapeutics. Health Quest Publications, 1997. Pp. 113-120.
- Lewin A et al. The effect of Coenzyme Q10 on sperm motility and function. 1997. Molec Aspects Med 18, Suppl: S213-S219.
- Miquel J et al. Mitochondrial role in cell aging. 1980. Exp Gerontol 15: 575-591.
- Miquel J. An update on the oxygen stress-mitochondrial mutation theory of aging: genetic and evolutionary implications. 1998. Exp Gerontol 33: 113-126.
- RICHIE JP JR. The role of GSH in aging and cancer. Experimental Gerontoloygy 27:615-626, 1992
- Satoh K et al. Lipophilic HMG-CoA reductase inhibitors increase myocardial stunning in dogs. 2000. J Cardiovasc Pharmacol 35: 256-62.
- Schardt F et al. Effect of coenzyme Q10 on ischaemia-induced ST-segment depression: a double blind, placebo-controlled crossover study. In Folkers K et al. (eds), Biomedical and Clinical Aspects of Coenzyme Q, vol. 5. Elsevier, 1986. Pp. 385-403.
- Senni M et al. Use of echocardiography in the management of congestive heart failure in the community. 1999. J Am Coll Cardiol 33: 164-170.
- Serra G et al. Evaluation of CoQ10 in patients with moderate heart failure and chronic stable effort angina. In Folkers K et al. (eds), Biomedical and Clinical Aspects of Coenzyme Q, vol. 6. Elsevier, 1991. Pp. 327-338.
- Shigenaga MK, Hagen TM, Ames BN. Oxidative damage and mitochondrial decay in aging. PNAS. 1994 Nov 8;91(23):10771-8.
- Singh RB et al. Effect of coenzyme Q10 on experimental atherosclerosis and chemical composition and quality of atheroma in rabbits. 2000. Atherosclerosis 148: 275-282.
- Singh RB et al. Randomized, double-blind placebo-controlled trial of coenzyme Q10 in patients with acute myocardial infarction. 1998. Cardiovasc Drugs Ther 12: 347-353.
- Soja AM et al. Treatment of congestive heart failure with coenzyme Q10 illuminated by meta-analyses of clinical
- Stocker R et al. Ubiquinol-10 protects human low density lipoprotein more efficiently against lipid peroxidation that does a-tocopherol. 1991. Proc Natl Acad Sci USA 88: 1646-1650.
- Thomas S et al. Cosupplementation with coenzyme Q prevents the prooxidant effect of a-tocopherol and increases the resistance of LDL to transition metal-dependent oxidation initiation. 1996. Arterioscler Thromb Vasc Biol 16: 687-696.
- Thomas S et al. Inhibition of LDL oxidation by ubiquinol-10. A protective mechanism for coenzyme Q in atherolsclerosis? 1997. Molec Aspects Med 18, Suppl: S85-S103.
- Thomas S et al. Oxidation and antioxidation of human low-density lipoprotein and plasma exposed to 3-morpholinosydnonimine and reagent peroxynitrite. 1998. Chem Res Toxicol 11: 484-494.
- Tresch DD. The clinical diagnosis of heart failure in older patients. 1997. J Am Geriatr Soc 45: 1128-1133. trials. 1997. Molec Aspects Med. 18, Suppl: S159-S168.
- Udompataikul, M. & all. "An oral nutraceutical containing antioxidants, minerals and glycosaminoglycans improves skin roughness and fine wrinkles." International Journal of Cosmetic Science. 2009.